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I have been taking 1600 mg for 35 years. It is the only medicine that my doctor tried that really worked to relieve my panic disorder. However, it is only currently being produced by one drug company, and they are really expensive. It is considered an "old fashioned" medicine. Right now my insurance is covering it with a $50 copay. I don't know of any other medicine that is similar, so I guess I'm stuck for now.
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Benzodiazepines, are way better. Valium or clonazepam have a VERY long half life.(last about 18-24hours) This medicines work great for me, for overall Anxiety. In my case Xanax is the only drug that helps with my Panic Attacks. It works the fastest. And that is very important, when you have a bad event, like Panic Attacks. Not Anxiety attacks, there is a big difference. Panic is a Event(Can last awhile).These can give you aftershocks after the initial event and initial dose of Xanax. Anxiety attacks don't last long, and are not near as bad. Panic Attacks can be up to 10 times worse, than anxiety attacks. No triggers needed for Panic, in my case. Anxiety attacks usually center around stress, difficult situations, etc.

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The first cases of myxedema were recognized in the mid-19th century (the 1870s), but its connection to the thyroid was not discovered until the 1880s when myxedema was observed in people following the removal of the thyroid gland (thyroidectomy).

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Because is intended as a general reference, this information may not include all possible uses, precautions, directions, reactions (including allergic), drug interactions, or withdrawal times.

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Goiter size declines very slowly, and some patients may need high-dose thyroid hormone (called suppressive thyroid therapy) for a short period.

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TPO-Ab and typical hypoechogenicity of the thyroid in high-resolution sonography at first presentation or during follow-up and low pertechnetate uptake in thyroid scintigraphy.

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TSH level signals that thyroid hormone levels are already high, and adding levothyroxine could cause them to increase to a dangerous level.

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In patients with nontoxic diffuse goiter or nodular thyroid disease, particularly the elderly or those with underlying cardiovascular disease, levothyroxine sodium therapy is contraindicated if the serum TSH level is already suppressed due to the risk of precipitating overt thyrotoxicosis.

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Levothyroxine may increase the therapeutic and toxic effects of both drugs, possibly due to increased receptor sensitivity to catecholamines.

The role of T 3 in cardiovascular disease is highlighted by NTIS in which the physiologic response to illnesses such as heart failure are characterized by a decrease in total and free T 3 with normal T 4 and TSH levels.

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There should be recognition that there are not enough data to resolve clinical disagreement among thyroid experts (called “clinical equipoise”) regarding treatment for hypothyroidism.

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Certain medications (such as cholestyramine, colestipol, colesevelam, antacids, sucralfate, simethicone, iron, sodium polystyrene sulfonate, calcium supplements, orlistat, sevelamer, among others) can decrease the amount of thyroid hormone that is absorbed by your body.

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